Step 1. Specify gene name(s):

Enter gene name(s) separated by "Return": OR Select TEXT file with list of ORF or gene names, one per line. WORD Documents do not work unless saved as TEXT:

Step 2. Choose expression change:

Get experiments where gene expression is -Select- Increased Decreased Increased or Decreased Not Changed Not Expressed/No Data by fold. (Enter a positive integer greater than or equal to 2)

Step 3.

Step 1. Choose expression change:

Get all genes that are -Select- Increased Decreased Increased or Decreased Not Changed Not Expressed/No Data by fold. (Enter a positive integer greater than or equal to 2)

Step 2. Select datasets you wish to query:

Effects of glycosylation defects on gene expression, University of Oregon Cullen PJ, et al. Evolution of expression during glucose limitation, Stanford University Ferea TL, et al. Exploration of essential gene functions via titratable promoter alleles, University of Toronto Mnaimneh S, et al. Expression analysis of snf/swi mutants, Harvard University, Stanford University Sudarsanam P, et al. Expression during sporulation, UCSF, Stanford University Chu S, et al. Expression during the cell cycle, Stanford University, Cold Spring Harbor Spellman PT, et al. The analysis in Spellman et al included data from Cho et al Expression during the diauxic shift, Stanford University DeRisi JL, et al. Expression during the unfolded protein response, UCSF Travers KJ, et al. Expression in response to DNA-damaging agents, Stanford University Gasch AP, et al. Expression in response to alpha-factor (over time), Rosetta Inpharmatics Roberts CJ, et al. Expression in response to alpha-factor (various concentrations), Rosetta Inpharmatics Roberts CJ, et al. Expression in response to anoxia (0 - 6 generations) and subsequent reoxygenation (6 - 7.6 generations) in galactose, University of Illinois, Urbana Lai LC, et al. Expression in response to anoxia (0 - 6 generations) and subsequent reoxygenation (6 - 7.6 generations) in glucose, University of Illinois, Urbana Lai LC, et al. Expression in response to arsenic (time and dose), NIEHS (NIH), UCSD, Stanford University Haugen AC, et al. Expression in response to environmental changes, Stanford University Gasch AP, et al. Expression in response to histone depletion, Stanford University Wyrick JJ, et al. Expression in response to peroxisome induction and repression, The Institute for Systems Biology and University of Alberta Smith JJ, et al. Expression in response to varying zinc levels, University of Missouri, Stanford University Lyons TJ, et al. Expression in the presence of small-molecule inhibitors of rapamycin (SMIRs), Harvard, Yale, UCLA Huang J, et al. Expression regulated by the PHO pathway, Stanford University Ogawa N, et al. Expression regulated by the calcineurin/Crz1 pathway, Stanford Yoshimoto H, et al. Gene regulation by HTZ1, SWR1 and SIR2, UCSF Meneghini MD, et al. Kobor MS, et al. Gene regulation by Ino80 and Arp8, FMI, Basel, Switzerland van Attikum H, et al. Gene regulation by Ino80 and Arp8 in response to MMS, FMI, Basel, Switzerland van Attikum H, et al. Gene regulation by Swr1, Htz1, and Ino80, National Cancer Institute Mizuguchi G, et al. Ploidy regulation of gene expression, Whitehead, MIT Galitski T, et al. Response to creating a single unrepaired DSB by HO endonuclease in nocodazole-arrested cells, Brandeis & Stanford Universities Lee SE, et al. Role of transcription factors in adaptation to arsenic-induced stress, NIEHS (NIH), UCSD, Stanford University Haugen AC, et al. Transition from fermentative to glycerol-based respiratory growth, Wayne State University School of Medicine Roberts GG and Hudson AP Select All

Step 3.